AOD-6904 5mg

 35,00

AOD9604 is a modified version of the hGH fragment 176-191 peptide (contains a di-sulfide bridge) and thus a derivative of human growth hormone (hGH). Originally developed as a lipolytic (fat burning) compound, AOD9604 has shown benefit in studies of heart disease, osteoarthritis/cartilage repair, and metabolic syndrome. AOD9604 stimulates lipolysis (the breakdown or destruction of fat) and inhibits lipogenesis in animal studies.

Delving into the distinct properties of AOD 9604, it stands out among other peptides. Comprising 15 amino acids from the C-terminus end of Growth Hormone Polypeptide (GH), AOD 9604 is unique in its impact on insulin levels and its dual action in stimulating lipolysis (fat breakdown) and inhibiting lipogenesis (fat accumulation). This sets it apart from other peptides that either focus on muscle growth or fat reduction.

In comparison with other common peptides like GHRP-6 and Ipamorelin, AOD 9604 uniquely does not impact insulin levels while actively promoting fat loss and preventing fat accumulation. Furthermore, studies indicate its safety for long-term use, highlighting its advantages over other peptides in terms of insulin impact, fat metabolism, and safety.

AOD 9604 is widely recognized for its weight loss benefits, owing to its manipulation of fat metabolism processes without affecting blood sugar levels or growth. Beyond this, it also contributes to muscle growth, making it appealing to bodybuilders and athletes, and has potential as a regenerative medicine product, aiding muscle recovery and cell regeneration. The peptide also shows promise in areas still under research, such as bone strengthening, anti-inflammatory properties for conditions like arthritis, and improved skin elasticity through collagen production stimulation.

 

For Research Use Only

 

Obesity drug codenamed AOD9604 highly successful in trials

Safety and Tolerability of the Hexadecapeptide AOD9604 in Humans

The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice

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